Full TGIF Record # 270950
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Web URL(s):http://www.newss.org/proceedings/Proceedings_NEPPSC16_Vol1.pdf#page=179
    Last checked: 03/15/2018
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Publication Type:
i
Proceedings
Content Type:Abstract or Summary Only
Author(s):Yu, J.; Raymer, P.
Author Affiliation:University of Georgia, Griffin, GA
Title:Resistance mechanisms to ACCase inhibitors in goosegrass from Georgia
Section:Turf and ornamentals
Other records with the "Turf and ornamentals" Section
Meeting Info.:Philadelphia, Pennsylvania: January 3-7, 2016
Source:Proceedings of the First Annual Meeting of the Northeastern Plant, Pest, and Soils Conference. 2016, p. 153.
Publishing Information:s.l.: Northeastern Weed Science Society; the Eastern Branch of the Entomological Society of America; the Northeastern Division of the American Phytopathological Society; the Northeast Branch of the American Society of Agronomy; the Crop Science Society of America; the Soil Science Society of America; and the American Society of Horticultural Science-Northeast Region
# of Pages:1
Keywords:TIC Keywords: Acetyl-CoA carboxylase inhibitors; Biotypes; Diclofop methyl; Eleusine indica; Herbicide efficacy; Herbicide resistance; Molecular genetics; Mutations; Resistance
Abstract/Contents:"A goosegrass biotype with suspected resistance to ACCase-inhibitors was identified in Georgia. The objectives of this research were to evaluate (1) the resistance level of this biotype to ACCase-inhibitors, (2) efficacy of various herbicide for control, and (3) the physiological and molecular basis of resistance. In greenhouse experiments, the rate of diclofop-methyl that reduced dry shoot biomass 50 percent (SR50) from the nontreated for the resistant (R) and susceptible (S)-biotypes measured 4100 and 221 g ai ha-1, respectively. The SR50 for sethoxydim measured 615 and 143 g ai ha-1 for the R and S-biotype, respectively. The R-biotype was cross-resistant to clethodim, fenoxaprop, and fluazifop. The R-biotype was equally susceptible to the S-biotype from foramsulfuron, glyphosate, MSMA, and topramezone, averaging 62 to 75 percent biomass reductions. In laboratory experiments, the two biotypes had similar foliar absorption of 14C-diclofop-methyl. Both biotypes metabolized 14C-diclofop-methyl to diclofop acid and a polar conjugate, but the R-biotype averaged ~2-times more degradation than the S-biotype. Gene sequencing revealed an Asp2078 to Gly substitution in the R-biotype that has previously conferred resistance to ACCase inhibitors. A second mutation was identified in the R-biotype that yielded a Thr1805 to Ser substitution that has not previously conferred ACCase-resistance in other species. "
Language:English
References:0
Note:This item is an abstract only!
ASA/CSSA/SSSA Citation (Crop Science-Like - may be incomplete):
Yu, J., and P. Raymer. 2016. Resistance mechanisms to ACCase inhibitors in goosegrass from Georgia. p. 153. In Proceedings of the First Annual Meeting of the Northeastern Plant, Pest, and Soils Conference. Philadelphia, Pennsylvania: January 3-7, 2016. s.l.: Northeastern Weed Science Society; the Eastern Branch of the Entomological Society of America; the Northeastern Division of the American Phytopathological Society; the Northeast Branch of the American Society of Agronomy; the Crop Science Society of America; the Soil Science Society of America; and the American Society of Horticultural Science-Northeast Region.
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http://www.newss.org/proceedings/Proceedings_NEPPSC16_Vol1.pdf#page=179
    Last checked: 03/15/2018
    Requires: PDF Reader
    Notes: Item is within a single large file
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