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| Publication Type:
| Report |
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| Content Type: | Abstract or Summary only |
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| Author(s): | Kelly, S. T.;
Coats, G. E. |
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| Author Affiliation: | Department of plant and Soil Sciences, Mississippi State University, Mississippi State, MS 39762-9555 |
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| Title: | Potential herbicide resistance in annual bluegrass to simazine |
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| Meeting Info.: | 51st Annual Meeting, Birmingham, AL, January 26-28, 1998 |
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| Source: | Southern Weed Science Society Proceedings. Vol. 51, January 1998, p. 90. |
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| Publishing Information: | Champaign, IL: Southern Weed Science Society. |
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| # of Pages: | 1 |
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| Keywords: | TIC Keywords: Poa annua; Simazine; Herbicide resistance; Atrazine; Diuron; Preventive maintenance
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| Abstract/Contents: | "Herbicide usage on fine turfgrass is relatively new, compared to usage on agronomic crops. Triazine resistant weed populations evolved in agronomic cropping situations about 20 years after continued usage. Suspected trazine resistant annual bluegrass (Poa annua L.) populations in fine turf are now reported, after about the same amount of time has passed since use on fine turf began. Two such reports were investigated, and resistance has been confirmed in Mississippi. At these locations (Meridian and Tupelo, MS) these biotypes were found to be more than 100 fold resistant to simazine, compared to a susceptible biotype. At each location, simazine had been used for more than 12 years; probably longer, though this was as far back as written records were kept. Further experimentation indicated these populations were cross resistant to atrazine as well, but not to diuron, which has the same mode of action but a different binding site on the D1 protein complex. These results are similar to those observed by other researchers investigating triazine-resistance in other weed species, and seem to point to a modified binding site on the D1 protein that the triazine herbicides bind; however, further experimentation will be conducted to confirm this. Triazine-resistance in these two populations almost certainly evolved because of exclusive use of a single herbicide for annual bluegrass control. Since resistance normally occurs as a result of a selection process, the most effective means of preventing resistance to a specific class of chemistry is by using herbicides with differing modes of action. This would delay, if not prevent, the increase or occurrence of a resistant biotype. If a population is thought to be resistant, keeping it isolated and controlling the spread of plant material to other areas is another means of deterring potential increases in the population. Monitoring the appearance or spread of escapes is another tactic to prevent localized spread of resistant biotypes. Spot spraying as needed to prevent seed development will further reduce the spread or recurrence of escapes the following year. Although herbicide resistance has the potential to develop in the turfgrass industry, the spread and speed of development can be averted by using appropriate management practices. Herbicides can continue to be an effective tool to control unwanted vegetation, but should be used in a fashion that will not contribute to increased populations of resistant biotypes." |
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| Language: | English |
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| References: | 0 |
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| Note: | Partial reprint appears in Mississippi Turfgrass Association Newsletter, 31(3) March 1998, p. 7
This item is an abstract only! |
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| ASA/CSSA/SSSA Citation (Crop Science-Like - may be incomplete):
Kelly, S. T., and G. E. Coats. 1998. Potential herbicide resistance in annual bluegrass to simazine. South. Weed Sci. Soc. Proc. 51:p. 90. |
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MSU catalog number: SB 611 .S6 v.51 |
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