Full TGIF Record # 336031
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Content Type:Abstract or summary only
Author(s):Rutland, Claudia A.; Gaines, Todd A.; Patterson, Eric L.; Cutti, Luan; Zeka, Brian; Llaca, Victor; Patel, Jinesh; Patel, Sejal; McElroy, Joseph S.
Author Affiliation:Rutland: Auburn University Department of Crop, Soil, and Environmental Sciences, Auburn, AL; Gaines: Colorado State University, Fort Collins, CO; Patterson: Michigan State University, East Lansing, MI; Cutti: Federal University of Rio Grande do Sul, Porto Alegre , Brazil; Zeka: Corteva, Johnston, IA; Llaca: Corteva Agriscience, Johnston, IA; Patel, J. and Patel, S.: Auburn University, Auburn, AL
Title:Unraveling the mechanism of quinclorac resistance in smooth crabgrass (Digitaria ischamemum)
Section:Oral student travel enrichment, experience and science policy update
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Meeting Info.:San Antonio, Texas: January 22 - 25, 2024
Source:Weed Science Society of America - Southern Weed Science Society Joint Meeting. 2024, p. 296.
Publishing Information:[United States]: Weed Science Society of America
# of Pages:1
Abstract/Contents:"Quinclorac is a unique synthetic auxin herbicide known for its grass-in-grass specificity, as other synthetic auxins only provide control over broadleaf species while leaving grasses unharmed. Thus, quinclorac is a useful turfgrass herbicide as it provides grass-in-grass control in turfgrass stands and also control of select broadleaf weeds. Resistance to quinclorac in smooth crabgrass has been previously identified, but the full mechanism of resistance is still unknown. Auxin functions as the regulator for the degradation of AUX/IAA proteins, and target-site mutations have been identified in other synthetic auxin herbicides in many of these AUX/IAAs. Research was conducted in order to determine if a specific AUX/IAA within smooth crabgrass has causative mutation which endows resistance to quinclorac. Herein we performed a comparative analysis of resistant and susceptible smooth crabgrass populations using Trinity and Trinotate to identify potential mutations within all copies of the AUX/IAA gene family. A novel genome was also produced, which allowed us to perform a differential gene expression (DGE) study to identify any other genes that may be up or down-regulated between the resistant and susceptible lines. A point mutation located outside the degron was identified with potential for conformational change within the active site of the AUX/IAA protein. DGE analysis is currently being conducted, but if the mutation is the causative mechanism, it is not expected to yield any biologically important differentially expressed genes. As of this writing, no metabolic genes of importance (Cytochrome P4050s, GSTs, ABC Transporters) have been identified as differential expressed"
Language:English
References:0
See Also:Original version appears in, ASA, CSSA, SSSA International Annual Meeting, 2023, p. 150531, with variant title, "Understanding the potential mechanisms of resistance of smooth crabgrass to quinclorac", R=333402. R=333402
Note:This item is an abstract only!
ASA/CSSA/SSSA Citation (Crop Science-Like - may be incomplete):
Rutland, C. A., T. A. Gaines, E. L. Patterson, L. Cutti, B. Zeka, V. Llaca, et al. 2024. Unraveling the mechanism of quinclorac resistance in smooth crabgrass (Digitaria ischamemum). Abstr. Annu. Meet. Weed Sci. Soc. Am. p. 296.
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https://wssa.net/wp-content/uploads/ilovepdf_merged-26.pdf#page=300
    Last checked: 04/18/2024
    Requires: PDF Reader
    Notes: Item is within a single large file
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